Background
Cardiac myosin-binding protein C (cMyC) is a novel biomarker of myocardial injury. We hypothesize that Calpain-mediated cleavage of cMyC is dependent on its phosphorylation status and can aid the differentiation between different types of myocardial injury
Conclusion
Phosphorylation and fragmentation pattern of cMyC are dependent on the type of myocardial injury and might aid the differentiation between Type-1 and non-Type-1 AMI